What I had in mind here was
paragraph 310 of the Catechism of the Catholic Church, which says, in part:
…with infinite wisdom and goodness God freely willed to create a world "in a state of journeying " toward its ultimate perfection. In God’s plan this process of becoming involves the appearance of certain beings and the disappearance of others, the existence of the more perfect alongside the less perfect, both constructive and destructive forces of nature. With physical good there exists also physical evil as long as creation has not reached perfection.
%between%
Nope, evolution is not a teleological force. So evolution strives for “perfection”? Any evidence for this profound assertion? “Evolution” occurs when one adapts to a more difficult niche, not when one becomes more reproductively efficient. If the latter is true, then
Eschericha coli is superior to
Homo sapiens. Returning to perfection, why is the termnial gene in the L-ascorbic acid biosynthesis pathway disabled in us? So many sailors died of scurvy before James Cook. Also, it stands to ask why meisois has not been perfected. Oh well, just look up Down syndrome, Kleinfelter syndrome, and Edwards syndrome on wikipedia about this.
Darwinian evolution is not egalitarian; it inhibits the proliferation of “unfit” phenotypes. For example, this explains why the frequence for the alleles for hereditary diseases are so low in human population. An example of this is cystic fibrosis, which occurs in about 1 in 25 people of Caucasian and Jewish descent. But it is also thought being heterozygous (having only one copy of the defective allele, which is a chloride ion (Cl-) transport gene is thought to confer an advantage under certain circumstances. One such circumstance is under the presence of cholera toxin, which is “intelligently designed,” by interfering with signal transduction by preventing the hydrolysis of GTP for the alpha unit of the g-protein, thus allowing phosphodiesterase to convert ATP to cAMP, causing the accumilation of that second messenger. (see
here)
Other hereditary diseases such as Lesch Nyhan syndrome (absence of a functional copy of the purine salvage enzyme hypoxanthine-guanine phosphoribosyltransferase), and galactosemia (which usually involves the absence of galactose 1-phosphate uridyltransferase, which transfers UMP (from 1-UDP-glucose) anomeric carbon of galactose. This sets up the next reaction to convert galactose into glucose, and then glucose-1-phopshate is converted to glucose-6-phosphate, so it could enter glycolysis, pentose phosphate pathway, or become stored as glycogen. (see
here)) These other diseases are recessive, like cystic fibrosis. If such diseases had such devastating effects on phenotype and are dominant, they would be weeded out. But diseases like Huntingtons’ disease are dominant, but they strike when one is past their reproductive age.
I guess evolution has “perfected” HIV’s (which causes AIDS, contrary to what Duesberg and Jonathan Wells’ opinions) ability to mimic a CD4 receptor in leukocytes, so it could enter the immune system. Also evolution has the potential to “perfect” the ability of reverse transciptase of rejecting azidothymidine (in its 5’ triphosphorylated form) as a substrate (AZT is a common medicine in anti-retroviral regiments).