O
o_mlly
Guest
I see nothing in the articles which support the narrative you have posted which is simply more evolutionary speculation. Staying within the data produced by the (repeatable) experiment evidences that no mutations were produced to enable penicillin-resistance in the population. No mutation means no evolution.Please read these references on the Lederberg Experiment and what it proves (especially the second one, which was written by Lederberg himself.)
I can see how you might think the idea of “mutations were always there” might support the idea that all genetic information for primates was present in the first bacteria. But it doesn’t actually support that strong a claim. All it does is support the claim that random mutations (Lederberg uses the word random) were there before exposure to an environment in which they proved beneficial. To borrow a word from the evolution denying lexicon, these are “micro-mutations”. They are small enough to have occurred randomly. But that does not mean that “macro-mutations” for primates were present in bacteria. To get from bacteria to primates takes many many mutations. The only reasonable way those mutations could be properly cascaded is for some sort of selection process that approves or rejects micro-mutations as they occur. Those were the mutations studied by Lederberg.
If you believe either article support your narrative, please cite and quote the relevant text. What I do read in both articles is a re-statement of the evidence supporting the notion that the gene pool already contained within the population of bacteria elements capable of resisting penicillin. (It is logically incorrect to label the penicillin-resistant bacteria as “mutants” since these same bacteria were present in the initial plates. No change, therefore, no mutations.)
“Replicas to agar containing bacteriophage or streptomycin showed that mutants of Escherichia coli resistant to these agents existed in clones on the initial plates of indifferent agar medium.”
“Thus, by using their replica-plating technique, the Lederbergs demonstrated the existence of streptomycin-resistant mutants in a population of bacteria prior to their exposure to the antibiotic. Their results, along with those of many other experiments, have shown that environmental stress does not direct or cause genetic changes; it simply selects rare preexisting mutations that result in phenotypes better adapted to the new environment.”